Focal Interest
Focal Interest

Fluoride Effects and Removal

External reference: https://openalex.org/T11969

  1. F6H8 altered human liver cell metabolism and produced a PFAS-like metabolite
    F6H8, an ophthalmic treatment for dry eye, produces PFAS-like metabolites and triggers broad metabolic disruption in human hepatocytes, raising long-term safety concerns.
  2. PFOS detected rapidly with a dual-recognition chemiresistive sensor
    Chemiresistive sensor with molecularly imprinted polymer and fluorine-fluorine interactions achieves 1.3 ng·L⁻¹ detection of PFOS in environmental samples with validated accuracy.
  3. Neurodegeneration, Oxidative Stress, NGF/TrkA/P75NTR, and PGE2 Dysregulation Induced by PFOS Single and Repeated Treatment: Partial Protection by T3 and Other Therapeutic Approaches
    PFOS induces neurodegeneration in basal forebrain cholinergic neurons through oxidative stress, PGE2 dysregulation, and NGF pathway disruption; T3 treatment offers partial neuroprotection.
  4. Sunlight-driven photocatalyst cleaves PFAS carbon–fluorine bonds
    Z-scheme photocatalyst CuInS2/BiOCl composite enables visible-light-driven degradation of PFAS through directed charge transfer, achieving 96% removal in sunlight-driven continuous-flow systems.
  5. PFOA-linked bladder cancer signals identified in transcriptomic analysis
    Study reveals molecular mechanisms linking PFOA exposure to bladder cancer through integrative toxicogenomics and gene expression analysis, identifying key transcriptional signatures for cancer.
  6. Certain nitrogen compounds improved PFOA electrochemical degradation
    Electrochemical degradation of PFOA enhanced by nitrogen-containing compounds with lone-pair electrons; mechanisms involve direct electron transfer and reactive nitrogen species generation.
  7. DOM forms PFAS nanoclusters and lowers phytotoxicity
    In situ AFM reveals DOM-induced PFAS nanoclustering reduces phytotoxicity through altered bioaccessibility, challenging molecular dispersal-based risk paradigms.